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Yocto Project Mega- Manual. Chapter 1. The Yocto Project Development Manual! Getting Started with the Yocto Project. Setting Up the Development Host to Use the Yocto Project. Setting Up a Native Linux Host. Setting Up to Use CROss Platform. S (CROPS). Linux, Mac, or Windows). Once you complete the setup instructions for your. Poky, Extensible SDK, and Toaster. Setting Up BSP Layers. Setting Up to Work on a Kernel. Getting Ready for Traditional Kernel Development. Working With Yocto Project Source Files. Accessing Source Repositories. Accessing Index of Releases. Using the Downloads Page. Cloning the poky Repository. Checking Out by Branch in Poky. Checking Out by Tag in Poky. Performing a Simple Build. The Yocto Project Open Source Development Environment. Setting Up a Team Yocto Project Development Environment.
Submitting a Defect Against the Yocto Project. Submitting a Change to the Yocto Project. Bit. Bake, Open. Embedded- Core, meta- yocto. This tutorial provides instructions for installing a Chef server on an Oracle Compute Cloud Services instance or virtual machine (VM). The Nutanix Bible - A detailed narrative of the Nutanix architecture, how the software and features work and how to leverage it for maximum performance.Using Scripts to Push a Change Upstream and Request a Pull. Using Email to Submit a Patch. Understanding and Creating Layers. Creating Your Own Layer. Best Practices to Follow When Creating Layers. Avoid Duplicating Include Files. Structure Your Layers. Other Recommendations. Making Sure Your Layer is Compatible With Yocto Project. Yocto Project Compatibility Program Application. Enabling Your Layer. Using . bbappend Files. Prioritizing Your Layer. Creating a General Layer Using the yocto- layer Script. Customizing Images Using local. Customizing Images Using Custom IMAGE. Customizing Images Using Custom . Files. Customizing Images Using Custom Package Groups. Customizing an Image Hostname. Writing a New Recipe. Locate or Automatically Create a Base Recipe. Locating and Using a Similar Recipe. Storing and Naming the Recipe. Understanding Recipe Syntax. Running a Build on the Recipe. Configuring the Recipe. Enabling System Services. Sharing Files Between Recipes. Properly Versioning Pre- Release Recipes. Post- Installation Scripts. Single . c File Package (Hello World!). Makefile- Based Package. Splitting an Application into Multiple Packages. Packaging Externally Produced Binaries. Following Recipe Style Guidelines. Adding a New Machine. Adding the Machine Configuration File. Adding a Kernel for the Machine. Adding a Formfactor Configuration File. Finding Temporary Source Code. Using Quilt in Your Workflow. Using a Development Shell. Using a Development Python Shell. Building Targets with Multiple Configurations. Working With Libraries. Including Static Library Files. Combining Multiple Versions of Library Files into One Image. Preparing to Use Multilib. Using Multilib. Additional Implementation Details. Installing Multiple Versions of the Same Library. Enabling GObject Introspection Support. Enabling the Generation of Introspection Data. Disabling the Generation of Introspection Data. Testing that Introspection Works in an Image. Creating Partitioned Images. Creating Partitioned Images. Using Open. Embedded Image Creator Wic to Generate Partitioned Images. Operational Modes. You can provide your own. Using an Existing Kickstart File. Generate an Image using an Existing Kickstart File. Using a Modified Kickstart File. Creating an Image Based on core- image- minimal and crownbay- noemgd. Using a Modified Kickstart File and Running in Raw Mode. Open. Embedded Kickstart (. Reference. Command: part or partition. Command: bootloader. Building an Initial RAM Filesystem (initramfs) Image. Configuring the Kernel. Creating a defconfig File. Creating Configuration Fragments. Fine- Tuning the Kernel Configuration File. Determining Hardware and Non- Hardware Features for the Kernel Configuration Audit Phase. Patching the Kernel. Create a Layer for your Changes. Finding the Kernel Source Code. Creating the Patch. Set Up Your Layer for the Build. Set Up for the Build. Build the Modified QEMU Kernel Image. Boot the Image and Verify Your Changes. Making Images More Secure. Creating Your Own Distribution. Creating a Custom Template Configuration Directory. Building a Tiny System. Goals and Guiding Principles. Understand What Contributes to Your Image Size. Trim the Root Filesystem. Remove Package Management Requirements. Look for Other Ways to Minimize Size. Iterate on the Process. Building Images for More than One Machine. Working with Packages. Excluding Packages from an Image. Incrementing a Package Version. Working With a PR Service. Manually Bumping PR. Automatically Incrementing a Package Version Number. Handling Optional Module Packaging. Making Sure the Packaging is Done. Must. include %s. Must. include %s. The default value of None. Can be a single string item or a list. Must include %s. Can be a single string item. Must. include %s. Must include %s. defaults to description if not set. Satisfying Dependencies. Using Runtime Package Management. Internet- facing). Build Considerations. Host or Server Machine Setup. Apache 2, Lighttpd, or Nginx), take the. Generating and Using Signed Packages. Signing RPM Packages. Processing Package Feeds. Testing Packages With ptest. Adding ptest to Your Build. Getting Your Package Ready. Working with Source Files. Setting up Effective Mirrors. Getting Source Files and Suppressing the Build. Building Software from an External Source. Selecting an Initialization Manager. Using systemd Exclusively. Using systemd for the Main Image and Using Sys. Vinit for the Rescue Image. Selecting a Device Manager. Using Persistent and Pre- Populated/dev. Using devtmpfs and a Device Manager. Using an External SCM. Creating a Read- Only Root Filesystem. Creating the Root Filesystem. Post- Installation Scripts. Areas With Write Access. Performing Automated Runtime Testing. Enabling Runtime Tests on QEMU. Enabling Runtime Tests on Hardware. Selecting Systemd- boot. Target. Serial Console Connection. Beagle. Bone. Target. Edge. Router. Target, and Grub. Target), you need to. TEST. Writing New Tests. Class Attributes. Qemu. Target, Simple. Remote, and. Systemd- boot. Target). Instance Attributes. Installing Packages in the DUT Without the Package Manager. Debugging With the GNU Project Debugger (GDB) Remotely. Debugging with the GNU Project Debugger (GDB) on the Target. Debugging Parallel Make Races. Reproducing the Error. Creating a Patch for the Fix. Testing the Build. Maintaining Open Source License Compliance During Your Product's Lifecycle. Providing the Source Code. Providing License Text. Providing Compilation Scripts and Source Code Modifications. Using the Quick EMUlator (QEMU). Switching Between Consoles. Removing the Splash Screen. Disabling the Cursor Grab. Running Under a Network File System (NFS) Server. The QEMU Emulator. Eclipse Yocto Plug- in. Latency. TOP, Power. TOP, Perf, System. Tap. and Lttng- ust) that also added to the development experience. SDK Development Model. Using the Extensible SDK. Why use the Extensible SDK and What is in It? Setting Up to Use the Extensible SDK. Y. Extracting SDK................................... Setting it up.. Running the Extensible SDK Environment Setup Script. Dependency Detection and Mapping. License Detection. Adding Makefile- Only Software. CC. CXX, and so forth) in a similar. SDK's. environment setup script. Adding Node. js Modules. Working With Recipes. Finding Logs and Work Files. Setting Configure Arguments. Sharing Files Between Recipes. Restoring the Target Device to its Original State. RPM or OPKG). Installing Additional Items Into the Extensible SDK. Updating the Extensible SDK. Creating a Derivative SDK With Additional Components. Using the Standard SDK. Why use the Standard SDK and What is in It? Y. Extracting SDK.................................... Setting it up.. done. SDK has been successfully set up and is ready to be used. Running the SDK Environment Setup Script. Using the SDK Toolchain Directly. Passing Host Options. Makefile- Based Projects. Developing Applications Using Eclipse. Workflow Using Eclipse. Working Within Eclipse. Setting Up the Neon Version of the Eclipse IDE. Installing the Neon Eclipse IDE. Configuring the Neon Eclipse IDE. Installing or Accessing the Neon Eclipse Yocto Plug- in. Installing the Pre- built Plug- in from the Yocto Project Eclipse Update Site. Installing the Plug- in Using the Latest Source Code. Configuring the Neon Eclipse Yocto Plug- in. Configuring the Cross- Compiler Options. Configuring the Target Options. Creating the Project. Building the Project. Starting QEMU in User- Space NFS Mode. Deploying and Debugging the Application. Board Support Packages (BSP) - Developer's Guide. Example Filesystem Layout. README. sources File. Pre- built User Binaries. Sato) and. minimal live images when the BSP tarball has been created and. Yocto Project website. Layer Configuration File. Hardware Configuration Options. Miscellaneous BSP- Specific Recipe Files. Display Support Files. Linux Kernel Configuration. Developing a Board Support Package (BSP). Requirements and Recommendations for Released BSPs. Released BSP Requirements. Released BSP Recommendations. Customizing a Recipe for a BSP. Creating a new BSP Layer Using the yocto- bsp Script. Managing Kernel Patches and Config Items with yocto- kernel. Introduction. Kernel Modification Workflow. Common Tasks. Creating and Preparing a Layer. Modifying an Existing Recipe. Creating the Append File. Applying Patches. Changing the Configuration. Using an Iterative Development Process. Generating Configuration Files. Modifying Source Code. Working With Your Own Sources. Working with Out- of- Tree Modules. Building Out- of- Tree Modules on the Target. Incorporating Out- of- Tree Modules. Inspecting Changes and Commits. What Changed in a Kernel? Showing a Particular Feature or Branch Change. Adding Recipe- Space Kernel Features. Working with Advanced Metadata. BSP Descriptions. Minnow Board). Organizing Your Source. Encapsulating Patches. Machine Branches. Feature Branches. SCC Description File Reference. Yocto Project Profiling and Tracing Manual. For example. EXTRA. Overall Architecture of the Linux Tracing and Profiling Tools. Basic Usage (with examples) for each of the Yocto Tracing Tools. The past couple of years have seen perf subsume a lot. Extrapolation suggests that at. Assistir The Vampire Diaries HD 7. Online. Na S. Damon (Ian Somerhalder) e Stefan Salvatore (Paul Wesley) s. Ao mesmo tempo, buscam resistir . 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Bleeding and liver disease Haemostatic defects § Impaired coagulation § Reduced synthesis of clotting proteins. Protein C, also known as autoprothrombin IIA and blood coagulation factor XIV,:6822 is a zymogen, the activated form of which plays an important role in regulating. Original Article. Tissue Plasminogen Activator for Acute Ischemic Stroke. The National Institute of Neurological Disorders and Stroke rt-PA Stroke Study Group*. Falsely. increased levels may occur if serum or plasma is not separated from the red. Substantial increases are secondarily (not genetically. Normal levels in the African American. Caucasian and Asian. Native Americans and Mexican Americans have lower normal levels. Caucasian and Asian populations. A prolonged a. PTT that normalizes immediately after mixing with. A prolonged a. PTT that remains prolonged. PTT that normalizes immediately. A prolonged PT that is corrected in the mixing study, along with a. PTT, suggests a factor VII deficiency. Test results are consistent.
II, V, or X (common pathway). PT is normalized after mixing studies and when a prolonged. PTT normalizes after mixing studies and remains normalized after. Interpretation of increased levels. Severely decreased or undetectable. The antigenic, but not the. PAI- 1 and a dysproteinemia. Similar to PAI- 1 antigen and activity levels, data regarding utility. G/5. G polymorphism in predicting venous thrombosis is conflicting. Read medical definition of Tissue plasminogen activator. Tissue plasminogen activator: (tPA) An enzyme that helps dissolve clots. It may be more useful when co- inherited with another thrombophilia marker. For example, Visanji et al found the risk for venous. G allele (4. G/4. G or 4. G/5. G genotypes) relative to that in patients. G/5. G genotype. 3. All patients. were heterozygous for factor V (Leiden) mutation and had experienced at. Furthermore, Zoller et al reported an. S deficiency who were homozygous for the 4. G allele. 3. 5. Plasminogen. Decreased levels are associated with liver disease, DIC, thrombolytic. Increased. levels are associated with trauma, infection, acute myocardial infarction. A functional assay is usually the. The antigen level is most often. Low values may be obtained in individuals on oral anticoagulant. Heparin levels up to 1 IU/m. L do not interfere with test results for. Protein C levels are significantly decreased in. Low levels in. both activity and antigen assays are suggestive of type I deficiency. II deficiency (dysproteinemia). A rare hereditary deficiency. Levels are significantly decreased in. Low levels. in both activity and antigen (free and total) assays are suggestive of type. I deficiency, whereas low activity in the presence of normal total antigen. II deficiency (dysproteinemia). Type III deficiency is. A rare hereditary deficiency can. Venous thrombosis risk. The combination may also lead to cerebral sinus and spinal vein thromboses. Elevated. levels are associated with an increased risk of thrombosis and found in. DIC, DVT, and PE. Levels are. also increased in individuals with antithrombin deficiency. Prothrombin Time (PT). Prolonged PT results suggest a potential bleeding disorder that may be. II, V, VII, or X. Prolonged results are. When patients taking. Results are. interpreted based on the international normalized ratio (INR). A reduction. of the a. PTT after the addition of hexagonal phase phospholipid is considered. The sensitivity and specificity of this test are 9. False- positives may be seen with specific factor inhibitors. VIII and V) and direct thrombin inhibitors. While false- negatives. LA is high, d. RVVT Screen with Reflex to. RVVT Confirm and d. RVVT 1: 1 Mix assay is suggested. TPA is inhibited by plasminogen activator. Elevated TPA levels are associated with an increased risk of. Deficiency of von. Willebrand factor cleaving protease can result in persistence of supranormal. Guidelines for the Early Management of Patients With Acute Ischemic Stroke. Intravenous rt. PAIntravenous fibrinolytic therapy for acute stroke is now widely accepted. The US FDA approved the use of intravenous rt. PA in 1. 99. 6, in part on the basis of the results of the 2- part NINDS rt. PA Stroke Trial, in which 6. PA (0. 9 mg/kg IV, maximum 9. In the first trial (Part I), the primary end point was neurological improvement at 2. NIHSS. In the second trial (Part II), the pivotal efficacy trial, the primary end point was a global OR for a favorable outcome, defined as complete or nearly complete neurological recovery 3 months after stroke. Treatment with intravenous rt. PA was associated with an increase in the odds of a favorable outcome (OR, 1. CI, 1. 2–2. 9). Excellent outcomes on individual functional measures were more frequent with intravenous rt. PA for global disability (4. The benefit was similar 1 year after stroke. The major risk of intravenous rt. PA treatment remains s. ICH. In the NINDS rt. PA Stroke Trial, early minimal neurological symptoms or neurological deterioration temporally associated with any intracranial hemorrhage occurred in 6. PA and 0. 6% of patients given placebo. However, mortality in the 2 treatment groups was similar at 3 months (1. Although the presence of edema or mass effect on baseline CT scan was associated with higher risk of s. ICH, patients with these findings were more likely to have an excellent outcome if they received fibrinolytic therapy. The presence of early ischemic changes on CT scan was not associated with adverse outcome. The likelihood of a favorable outcome also was associated with the severity of deficits and the patient’s age. Patients with mild to moderate strokes (NIHSS score < 2. The chances of a complete or nearly complete recovery among patients with severe stroke (NIHSS score of > 2. Four subsequent trials, the European Cooperative Acute Stroke Study (ECASS I and ECASS II) and the Alteplase Thrombolysis for Acute Noninterventional Therapy in Ischemic Stroke (ATLANTIS A and ATLANTIS B), enrolled subsets of patients in the . The NINDS investigators reported a time- to- treatment interaction in a subgroup analysis of the NINDS rt. PA Stroke Trial. 9. Treatment with intravenous rt. PA initiated within 9. OR of 2. 1. 1 (9. CI, 1. 3. 3–3. 5. In comparison, the OR for good outcome at 3 months for treatment with intravenous rt. PA initiated within 9. CI, 1. 0. 9–2. 6. The investigators concluded that the earlier that treatment is initiated, the better the result. A subsequent pooled analysis of all large, multicenter, placebo- controlled trials of intravenous rt. PA for acute stroke confirmed a time effect. Investigation of the early time epoch in the NINDS trials revealed a potential confounder in the original data: 1. PA between 9. 1 and 1. NIHSS score of < 5 compared with 4% of the placebo patients. On the basis of this observation, it has been suggested that the relative preponderance of mild strokes with a likely good outcome in the intravenous rt. PA treatment group may explain the entire benefit reported for patients treated between 9. Subsequent reanalysis showed that the imbalance in patients with minor stroke did not explain the difference between treatment and placebo. The adjusted OR for 3- month favorable outcome (ORs for treatment compared with placebo) for the subgroup of patients from the 2 NINDS intravenous rt. PA stroke trials with NIHSS score of < 5 at baseline and time from stroke onset to treatment of 9. Indeed, when all possible subgroups were examined separately, no effect of the severity imbalance could be shown to influence the overall result that intravenous rt. PA therapy positively influenced outcome. In separate analyses by independent groups, an identical finding was reached: Baseline imbalances in the numbers of patients with mild stroke did not explain the overall study result. Subsequent to the approval of intravenous rt. PA for treatment of patients with acute ischemic stroke, numerous groups reported on the utility of the treatment in a community setting. Some groups reported rates of intracranial hemorrhage and favorable outcomes that were similar to those found in the NINDS trials, but others did not. It is now clear that the risk of hemorrhage is proportional to the degree to which the NINDS protocol is not followed. In addition to the risk of s. ICH, other potential adverse experiences include systemic bleeding, myocardial rupture if fibrinolytics are given within a few days of acute myocardial infarction, and reactions such as anaphylaxis or angioedema, although these events are rare. Orolingual angioedema reactions (swelling of tongue, lips, or oropharynx) are typically mild, transient, and contralateral to the ischemic hemisphere. Angioedema is estimated to occur in 1. PA treatment for ischemic stroke. Risk of angioedema is associated with angiotensin- converting enzyme inhibitor use and with infarctions that involve the insular and frontal cortex. Empiric monitoring recommendations include inspection of tongue, lips, and oropharynx after intravenous rt. PA administration. Empiric treatment recommendations include intravenous ranitidine, diphenhydramine, and methylprednisolone. The largest community experience, the SITS- ISTR Registry (Safe Implementation of Thrombolysis in Stroke–International Stroke Thrombolysis Register, which incorporates the SITS- MOST . The approval was conditional on the completion of a prospective registry of patient treatment experience with intravenous rt. PA within the 3- hour window from stroke onset. SITS- ISTR reported on 1. The frequency of early neurological deterioration temporally associated with substantial parenchymal hematoma after intravenous rt. PA was 1. 6% (9. 5% CI, 1. The frequency of favorable outcome (combined m. RS scores of 0, 1, and 2) at 9. CI, 5. 5. 3%–5. 7. PA patients, comparable to the favorable outcome rate among patients treated within 3 hours in the pooled analysis of the 6 randomized trials. These findings appear to confirm the safety of intravenous rt. PA within the 3- hour window at sites that have an institutional commitment to acute stroke care. With > 1. 5 years of fibrinolytic experience in acute ischemic stroke, multiple groups have reported their outcomes in treating patients with “off- label” fibrinolysis. These groups report the use of fibrinolysis in patients with conditions including extreme age (> 8. Overall, the outcomes in the treated patients with these contraindications were better than nontreated “controls” from registry data. Rates of s. ICH were not increased in these reports. Because stroke patients continue to present with conditions not specifically stated in the original indications for and usage of intravenous rt. PA, further experience may allow consideration for fibrinolysis in these situations. Extended Window for Intravenous rt. PASubsequent to the NINDS trials, 5 clinical trials have tested the use of intravenous rt. PA up to 6 hours after stroke onset without specialized imaging for patient selection. The first 4 trials, ECASS I, ECASS II, ATLANTIS A, and ATLANTIS B,1. None of these 4 trials was individually positive on its prespecified primary end point. In a pooled individual patient- level analysis of these 4 trials, a benefit of therapy in the 3- to 4. OR, 1. 4. 0; 9. 5% CI, 1. Fibrinolytic therapy in the 4. OR, 1. 1. 5; 9. 5% CI, 0. In the 3- to 4. 5- hour window, across all trials, rates of radiological parenchymal hematoma were higher with fibrinolytic therapy, 5. In the 4. 5- to 6- hour window, fibrinolytic therapy increased rates of both radiological parenchymal hematoma (6. When data from all time windows in the first 6 large intravenous rt. PA trials were pooled, a time- to- treatment interaction was shown. Treatment with intravenous rt. PA initiated within 1. OR of 2. 8. 1 (9. CI, 1. 7. 5–4. 5. The OR for good outcome at 3 months for treatment with intravenous rt. PA initiated within 1. CI, 1. 1. 2–2. 1. CI, 1. 0. 5–1. 8. The ECASS III trial was undertaken to prove or disprove the benefit of intravenous rt. PA in the 3- to 4. In ECASS III, patients between 3. PA (n=4. 18) or placebo (n=4. The dosing regimen was 0. The inclusion and exclusion criteria for the trial were similar to those in the existing AHA Stroke Council guidelines for treatment of patients within 3 hours of stroke onset,1. NIHSS score > 2. INR was < 1. 7), and those who had the combination of a previous stroke and diabetes mellitus. Patients were permitted to receive low- dose parenteral anticoagulants for prophylaxis of DVT within 2. PA. Early neurological deterioration likely caused by intracranial hemorrhage was identified in 1. PA (2. 4%) and 1 subject administered placebo (0. OR, 9. 8. 5; 9. 5% CI, 1. P=0. 0. 08). 1. 69 However, mortality in the 2 treatment groups did not differ significantly and was nominally higher among the subjects treated with placebo. The primary efficacy outcome in ECASS III was excellent 9. RS global disability scale (m. RS score 0–1). This outcome was more frequent with intravenous rt. PA (5. 2. 4%) than placebo (4. OR, 1. 3. 4; 9. 5% CI, 1. CI, 1. 0. 1–1. 3. P=0. 0. 4). The ECASS III findings align with preclinical and clinical data that suggest a time dependency for benefit from treatment with intravenous rt. PA. The point estimate for the degree of benefit seen in ECASS III (OR for global favorable outcome, 1. CI, 1. 0. 0–1. 6. NINDS study (OR, 1. CI, 1. 2–2. 9)1. 66,1. PA (OR, 1. 4). 9. Overall, the ECASS III results were consistent with the results of previous trials,9. PA can be given safely to, and can improve outcomes for, carefully selected patients treated 3 to 4. In June 2. 01. 2, the results from the Third International Stroke Trial (IST- 3), the largest randomized, placebo- controlled trial to date of intravenous rt. PA, were published. The trial enrolled 3. L/kg in the active arm. Eligibility criteria were similar to other intravenous rt. |